ATP disodium ntsev CAS 987-65-5

CAS:987-65-5

Kev Huv Si:99%

Cov mis molecular:C10H17N5NaO13P3

Qhov Hnyav Molecular:531.18

EINECS:213-579-1

Lub Sijhawm Khaws Cia:2 xyoos

Cov lus txhais:ADENOSINETRIPHOSPHATEDISODIUM; ADENOSINETRIPHOSPHATE, DISODIUMSALT; ADENYLPYROPHOSPHORICACIDDISODIUMSALT; ADENOSINE-5′-TRIPHOSPHATEHYDRATEDISODIUMSALT; ADENOSINE-5′-TRIPHOSPHATENA2-NTSEV; ADENOSINE-5′-TRIPHOSPHORICACID, DISODIUM; ADENOSINE-5′-TRIPHOSPHORICACIDDISODIUMDIHYDROGENSALT; ADENOSINE5′-TRIPHOSPHORICACIDDISODIUMSALT

Sau Koj Cov Lus

Cov Khoom Qhia Txog Khoom

Rub tawm

Cov Cim Npe Khoom

ATP disodium ntsev CAS 987-65-5 yog dab tsi?

ATP disodium ntsev yog ib qho metabolite ntawm adenosine, ib qho multifunctional nucleoside triphosphate uas siv ua coenzyme rau kev hloov pauv lub zog hauv cov cell. Nws thauj cov zog tshuaj hauv cov cell rau kev ua metabolism. ATP disodium ntsev yog siv los ua ribose-modified deoxyadenosine diphosphate analogs ua P2Y1 receptor ligands.

Cov Lus Qhia Tshwj Xeeb

Qhov tsos	Hmoov dawb los yog hmoov dawb los yog hmoov siv lead ua, hygroscopic.
Qhov loj ntawm cov khoom me me	> 95% dhau los ntawm 80 mesh.
pH	2.5 ~ 3.5
Kev yaj tau	Yaj hauv dej; tsis yaj hauv ethanol & ether.
Oder	Tsis muaj ntxhiab
Cov dej ntsiab lus	6.0% ~ 12.0%
Chloride	≤0.05%
ntsev hlau	≤0.001%
Cov hlau hnyav	≤0.001%
Cov hlau lead	≤2.0ppm
Arsenic	≤1.0ppm

Daim Ntawv Thov

1. Kev siv rau kev tshawb fawb thiab kev lag luam
(1) Cov khoom siv ua kom zoo nkauj: ntxiv rau cov khoom siv tiv thaiv kev laus ntawm daim tawv nqaij, nws tuaj yeem txhim kho daim tawv nqaij elasticity thiab glossiness los ntawm kev ua kom lub zog ntawm lub hlwb metabolism.
(2) Cov khoom noj khoom haus ntxiv: siv los ua cov khoom noj khoom haus zoo hauv cov dej haus kis las thiab cov khoom noj muaj txiaj ntsig kom rov ua kom muaj zog sai.
2. Cov chaw kho mob
(1) Cov kab mob ntawm lub plawv: siv rau kev ua tsis tau zoo ntawm lub plawv, mob myocarditis, mob myocardial infarction thiab mob cerebral arteriosclerosis, thiab lwm yam, los ntawm kev txhim kho lub zog ntawm lub plawv thiab ua kom cov hlab ntsha hauv lub plawv nthuav dav (ua kom cov ntshav ntws ntau ntxiv li 30%), txo cov tsos mob ntawm lub plawv ischemia. Piv txwv li, rau cov neeg mob uas muaj mob myocardial infarction, ATP disodium tuaj yeem ua rau lub sijhawm poob ntawm ST segment luv dua thiab txo qhov siab tshaj plaws ntawm myocardial enzyme spectrum.
(2) Cov kab mob ntawm lub paj hlwb: kev kho mob pab rau cov teeb meem ntawm kev ntshav hauv lub hlwb, kev puas tsuaj rau lub hlwb thiab cov leeg nqaij qaug zog zuj zus, los ntawm kev nkag mus rau hauv cov ntshav-lub hlwb thaiv (permeability yog li 65%), txhawb kev kho cov hlwb hlwb thiab cov txheej txheem rov ua dua tshiab, thiab txhim kho qhov ceev ntawm cov hlab ntsha.
(3) Cov kab mob metabolic: Hauv kev kho mob hepatitis thiab cirrhosis, ATP disodium tuaj yeem txhim kho cov haujlwm mitochondrial ntawm hepatocytes, ua kom cov hepatocyte kho sai dua, thiab txo cov ALT thiab AST; nws kuj muaj cov txiaj ntsig zoo rau cov teeb meem ntshav qab zib (xws li peripheral neuropathy).
3. Cov cheeb tsam thov tshiab
(1) Lub tshuab xa tshuaj rau lub hom phiaj: ATP disodium tuaj yeem siv ua lub tshuab hloov kho, ua ke nrog liposomes lossis nanoparticles, kom ua tiav kev xa tshuaj rau lub hom phiaj los ntawm kev kho mob endocytosis. Piv txwv li, hauv kev kho mob qog noj ntshav, ATP-modified nanomedicines tuaj yeem txhim kho kev tua tshuaj ntawm cov tshuaj chemotherapy rau cov hlwb qog noj ntshav.
(2) Kev cog qoob loo ntawm tes thiab cov tshuaj biopharmaceuticals: Ua ib qho tseem ceeb ntawm cov khoom siv cog qoob loo ntawm tes, ATP disodium tuaj yeem txhawb kev loj hlob thiab kev qhia tawm cov protein ntawm CHO hlwb, HEK293 hlwb, thiab lwm yam, thiab siv dav hauv kev tsim cov tshuaj monoclonal antibody.