ATP Disodium ntsev CAS 987-65-5

CAS:987-65-5 : kuv

Purity:99%

Molecular Formula:C10H17N5NaO13P3

Molecular Luj:531.18 ib

EINECS:213-579-1 ib

Lub Sijhawm Cia:2 xyoo

Synonyms:ADENOSINETRIPHOSPHATEDISODIUM; ADENOSINETRIPHOSPHATE, DISODIUMSALT; ADENYLPYROPHOSPHORICACIDDISODIUMSALT; ADENOSINE-5′-TRIPHOSPHATEHYDRATEDISODIUMSALT; ADENOSINE-5′-TRIPHOSPHATENA2- ntsev; ADENOSINE-5'-TRIPHOSPHORICACID, DISODIUM; ADENOSINE-5′-TRIPHOSPHORICACIDDISODIUMDIHYDROGENSALT; ADENOSINE5'-TRIPHOSPHORICACIDDISODIUMSALT

Tso Koj Cov Lus

Product Detail

Download tau

Khoom cim npe

ATP disodium ntsev CAS 987-65-5 yog dab tsi?

ATP disodium ntsev yog ib qho metabolite ntawm adenosine, multifunctional nucleoside triphosphate uas yog siv los ua coenzyme rau intracellular zog hloov hauv hlwb. Nws thauj tshuaj lub zog hauv cov hlwb rau cov metabolism. ATP disodium ntsev yog siv los tsim cov ribose-hloov deoxyadenosine diphosphate analogs li P2Y1 receptor ligands.

Specification

Qhov tshwm sim	Dawb hmoov los yog tawm- dawb hmoov los yog siv lead ua hmoov, hygroscopic.
Particle loj	≥ 95% dhau los ntawm 80 mesh.
pH	2.5 ~ 3.5 hli
Solubility	Soluble hauv dej; insoluble hauv ethanol & ether.
Oder	Oderless
Cov ntsiab lus dej	6.0% ~ 12.0%.
Chloride	≤ 0.05%
Hlau ntsev	≤ 0.001%
Cov hlau hnyav	≤ 0.001%
Coj	≤ 2.0ppm
Arsenic	≤ 1.0ppm

Daim ntawv thov

1. Kev lag luam thiab kev tshawb fawb siv
(1) Cov khoom siv tshuaj pleev ib ce: ntxiv rau cov khoom siv tiv thaiv kev laus ntawm daim tawv nqaij, nws tuaj yeem txhim kho cov tawv nqaij elasticity thiab glossiness los ntawm kev ua kom lub zog ntawm tes metabolism.
(2) Khoom noj khoom haus additives: siv los ua cov khoom noj khoom haus ntxiv rau hauv cov dej haus ua si thiab cov khoom noj ua haujlwm kom rov ua kom lub zog sai sai.
2. Cov chaw kho mob
(1) Cov kab mob plawv: siv rau lub plawv tsis ua hauj lwm, myocarditis, myocardial infarction thiab cerebral arteriosclerosis, thiab lwm yam, los ntawm kev txhim kho myocardial zog metabolism thiab dilating coronary hlab ntsha (ua kom ntshav txaus li ntawm 30%), relieving myocardial ischemia. Piv txwv li, nyob rau hauv cov neeg mob uas mob myocardial infarction, ATP disodium tuaj yeem ua rau luv luv ST ntu lub caij nplooj zeeg thiab txo tus nqi siab tshaj plaws ntawm myocardial enzyme spectrum.
(2) Cov kab mob hauv lub paj hlwb: pab kho cov sequelae ntawm cerebral hemorrhage, lub hlwb puas thiab cov leeg nqaij atrophy, los ntawm kev nkag mus rau cov ntshav-hlwb barrier (permeability yog li ntawm 65%), txhawb cov hlab ntsha cell membrane kho thiab paj hlwb txheej txheem, thiab txhim kho cov hlab ntsha conduction tshaj tawm.
(3) Cov kab mob metabolic: Hauv kev kho kab mob siab thiab mob cirrhosis, ATP disodium tuaj yeem txhim kho cov haujlwm mitochondrial ntawm hepatocytes, ua kom cov hepatocytes kho, thiab txo cov qib ALT thiab AST; Nws kuj muaj kev txhawb nqa kev txhim kho ntawm cov teeb meem ntshav qab zib (xws li peripheral neuropathy).
3. Cov chaw thov uas tshwm sim
(1) Lub hom phiaj kev xa tshuaj: ATP disodium tuaj yeem siv los ua tus neeg nqa khoom hloov pauv, ua ke nrog liposomes lossis nanoparticles, kom ua tiav cov tshuaj xa mus los ntawm receptor-mediated endocytosis. Piv txwv li, hauv kev kho mob qog, ATP-hloov nanomedicines tuaj yeem txhim kho kev xaiv tua cov tshuaj tua kab mob ntawm cov qog nqaij hlav cancer.
(2) Cell kab lis kev cai thiab biopharmaceuticals: Raws li ib feem tseem ceeb ntawm cell kab lis kev cai nruab nrab, ATP disodium tuaj yeem txhawb txoj kev loj hlob thiab kev qhia protein ntawm CHO hlwb, HEK293 hlwb, thiab lwm yam, thiab yog siv dav hauv kev tsim cov tshuaj monoclonal antibody.